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Apoptosis-derived membrane vesicles drive the cGAS–STING pathway and enhance type I IFN production in systemic lupus erythematosus

Crossref DOI link: https://doi.org/10.1136/annrheumdis-2018-212988

Published Print: 2018-10

Update policy: https://doi.org/10.1016/elsevier_cm_policy

Authors

Kato, Yasuhiro

Park, JeongHoon

Takamatsu, Hyota

Konaka, Hachirou

Aoki, Wataru

Aburaya, Syunsuke

Ueda, Mitsuyoshi

Nishide, Masayuki

Koyama, Shohei

Hayama, Yoshitomo

Kinehara, Yuhei

Hirano, Toru

Shima, Yoshihito

Narazaki, Masashi

Kumanogoh, Atsushi
Funding

Funding for this research was provided by:

Naito Foundation

Core Research for Evolutional Science and Technology

Ministry of Education, Culture, Sports, Science and Technology

Japan Agency for Medical Research and Development

Japan Society for the Promotion of Science

Center of Innovation Program
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Text and Data Mining valid from 2018-10-01

Text and Data Mining valid from 2018-10-01

Version of Record valid from 2018-06-26
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This article is maintained by: Elsevier

Article Title: Apoptosis-derived membrane vesicles drive the cGAS–STING pathway and enhance type I IFN production in systemic lupus erythematosus

Journal Title: Annals of the Rheumatic Diseases

CrossRef DOI link to publisher maintained version: https://doi.org/10.1136/annrheumdis-2018-212988

Content Type: article

Copyright: Copyright © 2018 © Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2018.No commercial use is permitted unless otherwise expressly granted. Published by Elsevier B.V.