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PKM2-dependent metabolic reprogramming in CD4+ T cells is crucial for hyperhomocysteinemia-accelerated atherosclerosis

Crossref DOI link: https://doi.org/10.1007/s00109-018-1645-6

Published Online: 2018-05-07

Published Print: 2018-06

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Lü, Silin

Deng, Jiacheng

Liu, Huiying

Liu, Bo

Yang, Juan

Miao, Yutong

Li, Jing

Wang, Nan

Jiang, Changtao

Xu, Qingbo

Wang, Xian

Feng, Juan
Funding

Funding for this research was provided by:

National Natural Science Foundation of P. R. China (91439206)

National Natural Science Foundation of P. R. China (81770445)

National Key Research and Development Program of China (2016YFC0903100)

Special Program of Peking University for Clinical Medicine + X (PKU2017LCX09)
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Article History

Received: 15 August 2017

Revised: 10 April 2018

Accepted: 29 April 2018

First Online: 7 May 2018

Compliance with ethical standards

: All animal procedures were approved by the Committee on the Ethics of Animal Experiments of the Health Science Center of Peking University, and the investigation conformed to the Guide for the Care and Use of Laboratory Animals published by the US National Institutes of Health (NIH Publication, 8th Edition, 2011).

: The authors declare that they have no conflict of interest.