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METTL14 derived from exosomes of M1 macrophages promotes high glucose-induced apoptosis, inflammation and oxidative stress in glomerular endothelial cells by mediating PAQR3 m6A modification

Crossref DOI link: https://doi.org/10.1007/s10157-024-02536-0

Published Online: 2024-07-30

Published Print: 2024-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

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