Loss of COMMD1 and copper overload disrupt zinc homeostasis and influence an autism-associated pathway at glutamatergic synapses
Crossref DOI link: https://doi.org/10.1007/s10534-014-9764-1
Published Online: 2014-07-10
Published Print: 2014-08
Update policy: https://doi.org/10.1007/springer_crossmark_policy
Baecker, Tanja
Mangus, Katharina
Pfaender, Stefanie
Chhabra, Resham
Boeckers, Tobias M.
Grabrucker, Andreas M.
Text and Data Mining valid from 2014-07-10