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Mutation of Beclin1 acetylation site at K414 alleviates high glucose-induced podocyte impairment in the early stage of diabetic nephropathy by inhibiting hyperactivated autophagy

Crossref DOI link: https://doi.org/10.1007/s11033-022-07242-2

Published Online: 2022-02-17

Published Print: 2022-05

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Xu, Jun https://orcid.org/0000-0001-7266-244X
Deng, Yujie https://orcid.org/0000-0001-6349-2215
Ke, Yingying https://orcid.org/0000-0002-8575-5108
Zhu, Yunxia https://orcid.org/0000-0002-8695-7545
Wang, Ping https://orcid.org/0000-0003-4109-5600
Yu, Qing

Li, Chengqian

Shi, Bimin https://orcid.org/0000-0002-7032-5367
Funding

Funding for this research was provided by:

National Natural Science Foundation of China (81603476, 81600684)

Youth Fund Project of Shandong Natural Science Foundation (ZR2016HQ16)
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Text and Data Mining valid from 2022-02-17

Version of Record valid from 2022-02-17
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Article History

Received: 24 November 2021

Accepted: 8 February 2022

First Online: 17 February 2022

Declarations

:

: The authors declare that they have no competing interests.

: The experimental protocol was approved by the Ethics Committee for Animal Experimentation of The Affiliated Hospital of Qingdao University.

: Not applicable.

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