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Deficiency of RAB39B Activates ER Stress-Induced Pro-apoptotic Pathway and Causes Mitochondrial Dysfunction and Oxidative Stress in Dopaminergic Neurons by Impairing Autophagy and Upregulating α-Synuclein

Crossref DOI link: https://doi.org/10.1007/s12035-023-03238-6

Published Online: 2023-01-30

Published Print: 2023-05

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Chiu, Ching-Chi

Weng, Yi-Hsin

Yeh, Tu-Hsueh

Lu, Juu-Chin

Chen, Wan-Shia

Li, Allen Han-Ren

Chen, Ying-Ling

Wei, Kuo-Chen

Wang, Hung-Li https://orcid.org/0000-0002-4328-0685
Funding

Funding for this research was provided by:

Ministry of Science and Technology, Taiwan (MOST109-2314-B-182-081-MY3, MOST111-2326-B-182-001-MY3)

Chang Gung Medical Foundation (CMRPD1H0282, CMRPD1H0283, CMRPG3J0763, CMRPG3K0952)

Chang Gung University (EMRPD1M0451, UMRPD1M003)
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Text and Data Mining valid from 2023-01-30

Version of Record valid from 2023-01-30
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Article History

Received: 21 May 2022

Accepted: 17 January 2023

First Online: 30 January 2023

Declarations

:

: Not applicable. This is an observational study. The Chang Gung University Research Ethics Committee has confirmed that no ethical approval is required.

: Not applicable.

: The authors declare no competing interests.

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