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Galantamine Inhibits Aβ1–42-Induced Neurotoxicity by Enhancing α7nAChR Expression as a Cargo Carrier for LC3 Binding and Aβ1–42 Engulfment During Autophagic Degradation

Crossref DOI link: https://doi.org/10.1007/s13311-019-00803-7

Published Print: 2020-04

Update policy: https://doi.org/10.1016/elsevier_cm_policy

Authors

Lin, Ming-Wei

Chen, Yi-Hung

Yang, Han-Ben

Lin, Chi Chien

Hung, Shih-Ya https://orcid.org/0000-0003-0749-1562
Funding

Funding for this research was provided by:

the Ministry of Science and Technology of Taiwan (MOST 108-2320-B-039-029-MY3)

China Medical University Hospital (DM-106-131)

the iEGG and Animal Biotechnology Center in The Feature Areas Research Center Program within the framework of the Higher Education Sprout Project led by the Ministry of Education (MOE-107-S-0023-E)
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Text and Data Mining valid from 2020-04-01

Version of Record valid from 2023-08-18
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This article is maintained by: Elsevier

Article Title: Galantamine Inhibits Aβ1–42-Induced Neurotoxicity by Enhancing α7nAChR Expression as a Cargo Carrier for LC3 Binding and Aβ1–42 Engulfment During Autophagic Degradation

Journal Title: Neurotherapeutics

CrossRef DOI link to publisher maintained version: https://doi.org/10.1007/s13311-019-00803-7

Content Type: article

Copyright: Copyright © 2020 The American Society for Experimental NeuroTherapeutics, Inc. Published by Elsevier B.V. All rights reserved.

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