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Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival

Crossref DOI link: https://doi.org/10.1038/s41374-018-0094-8

Published Print: 2019-03

Update policy: https://doi.org/10.1016/elsevier_cm_policy

Authors

Lee, Grace Sanghee

Salazar, Hector F.

Joseph, Giji

Lok, Zoe Shin Yee

Caroti, Courtney M.

Weiss, Daiana

Taylor, W. Robert

Lyle, Alicia N.
Funding

Funding for this research was provided by:

U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute (5T32HL007745-24)

U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute (F32HL137401)

U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute (P01HL095070)

U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute (R00HL119567)
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Text and Data Mining valid from 2019-03-01

Text and Data Mining valid from 2019-03-01

Version of Record valid from 2023-01-11
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This article is maintained by: Elsevier

Article Title: Osteopontin isoforms differentially promote arteriogenesis in response to ischemia via macrophage accumulation and survival

Journal Title: Laboratory Investigation

CrossRef DOI link to publisher maintained version: https://doi.org/10.1038/s41374-018-0094-8

Content Type: article

Copyright: © 2018 United States & Canadian Academy of Pathology.