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Stem cell persistence in CML is mediated by extrinsically activated JAK1-STAT3 signaling

Crossref DOI link: https://doi.org/10.1038/s41375-019-0427-7

Published Online: 2019-03-06

Published Print: 2019-08

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Kuepper, Maja Kim

Bütow, Marlena

Herrmann, Oliver

Ziemons, Janine

Chatain, Nicolas https://orcid.org/0000-0003-4485-3120
Maurer, Angela

Kirschner, Martin

Maié, Tiago

Costa, Ivan G. https://orcid.org/0000-0003-2890-8697
Eschweiler, Jörg

Koschmieder, Steffen https://orcid.org/0000-0002-1011-8171
Brümmendorf, Tim H.

Müller-Newen, Gerhard https://orcid.org/0000-0002-0820-7693
Schemionek, Mirle
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Article History

Received: 9 July 2018

Revised: 14 December 2018

Accepted: 2 January 2019

First Online: 6 March 2019

Compliance with ethical standards

:

: Steffen Koschmieder reports having served on the advisory boards for Pfizer, Incyte/Ariad, Novartis, AOP, BMS, and CTI; honoraria: Novartis, BMS, Pfizer, Incyte/Ariad, Shire, Janssen; scientific research support: Novartis Foundation, BMS, Novartis; Others: Alexion. The remaining authors declare that they have no conflict of interest.