Crossmark

AKT inhibition impairs PCNA ubiquitylation and triggers synthetic lethality in homologous recombination-deficient cells submitted to replication stress

Crossref DOI link: https://doi.org/10.1038/s41388-019-0724-7

Published Online: 2019-01-31

Published Print: 2019-05

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Villafañez, Florencia

García, Iris Alejandra

Carbajosa, Sofia

Pansa, María Florencia

Mansilla, Sabrina

Llorens, María Candelaria

Angiolini, Virginia

Guantay, Laura

Jacobs, Heinz

Madauss, Kevin P.

Gloger, Israel

Gottifredi, Vanesa

Bocco, Jose Luis

Soria, Gaston
Funding

Funding for this research was provided by:

Ministry of Science, Technology and Productive Innovation, Argentina | National Agency for Science and Technology, Argentina | Fondo para la Investigación Científica y Tecnológica (PICT startup 2013-1553, PAE GSK 2014-0005, PAE-GSK 2014-0005)

GlaxoSmithKline (PAE GSK 2014-0005)

Consejo Nacional de Investigaciones Científicas y Técnicas

Instituto Nacional del Cáncer

Ministry Of Health, Argentina | Instituto Nacional del Cáncer

KWF Kankerbestrijding (10796/2016-2, 10032/2016-1)
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Article History

Received: 12 July 2018

Revised: 12 December 2018

Accepted: 3 January 2019

First Online: 31 January 2019

Compliance with ethical standards

:

: The authors declare that they have no conflict of interest.

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