Crossmark

TRIM24-mediated K27-linked ubiquitination of ULK1 alleviates energy stress-induced autophagy and promote prostate cancer growth in the context of SPOP mutation

Crossref DOI link: https://doi.org/10.1038/s41418-025-01582-9

Published Online: 2025-09-20

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Chen, Shimin

Lin, Jichun

Yang, Zhan https://orcid.org/0000-0001-7191-2910
Wang, Yuanjing

Wang, Qiang

Wang, Dong

Qu, Yue

Lin, Qian

Liu, Jia

Yan, Shi

Wang, Zixin

Qian, Xueyu

Xiao, Yutian https://orcid.org/0000-0002-3039-8342
Li, Xue

Chen, Yinuo

Fang, Wenshuo

Zhao, Jiaojiao

Lu, Zhimin https://orcid.org/0000-0002-2859-2736
Ren, He

Zhu, Yasheng

Ma, Leina https://orcid.org/0000-0003-3964-632X
Funding

Funding for this research was provided by:

National Natural Science Foundation of China (82372806, 82188102, 82030074, 82125026)

Natural Science Foundation of Shandong Province (ZR2021MC039)

Taishan Scholar Foundation of Shandong Province (tsqn202312378, tsqn202312378)

Ministry of Science and Technology of the People's Republic of China (2020YFA0803300)

Science and Technology Commission of Shanghai Municipality (22Y11905200)
License Information

Text and Data Mining valid from 2025-09-20

Version of Record valid from 2025-09-20
More Information

Article History

Received: 1 October 2024

Revised: 5 August 2025

Accepted: 10 September 2025

First Online: 20 September 2025

Competing interests

: The authors declare no competing interests.

: The studies involving human participants were reviewed and approved by the Institute Review Board of Shanghai Changhai Hospital. Written informed consent for participation was not required for this study in accordance with the national legislation and the institutional requirements.

Document is current

Any future updates will be listed below