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An IRAK1–PIN1 signalling axis drives intrinsic tumour resistance to radiation therapy

Crossref DOI link: https://doi.org/10.1038/s41556-018-0260-7

Published Online: 2019-01-21

Published Print: 2019-02

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Liu, Peter H. https://orcid.org/0000-0001-5037-085X
Shah, Richa B.

Li, Yuanyuan

Arora, Arshi

Ung, Peter Man-Un https://orcid.org/0000-0002-6849-5252
Raman, Renuka

Gorbatenko, Andrej

Kozono, Shingo

Zhou, Xiao Zhen

Brechin, Vincent

Barbaro, John M.

Thompson, Ruth

White, Richard M. https://orcid.org/0000-0001-9099-9169
Aguirre-Ghiso, Julio A.

Heymach, John V.

Lu, Kun Ping

Silva, Jose M.

Panageas, Katherine S.

Schlessinger, Avner https://orcid.org/0000-0003-4007-7814
Maki, Robert G.

Skinner, Heath D. https://orcid.org/0000-0003-1836-151X
de Stanchina, Elisa

Sidi, Samuel https://orcid.org/0000-0001-7649-0673
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Article History

Received: 6 June 2018

Accepted: 7 December 2018

First Online: 21 January 2019

Change Date: 1 February 2019

Change Type: Update

Change Details: In the sentence beginning “Moreover, IRAK1 inhibitors...” in the Abstract of the version of this Article originally published, it was unclear whether the present study shows synergy between IRAK1 and PIN1 inhibitors in response to pathogens or ionizing radiation. The sentence has been modified to clarify this.

Competing interests

: The authors declare no competing interests.