Crossmark

CLCN2 chloride channel mutations in familial hyperaldosteronism type II

Crossref DOI link: https://doi.org/10.1038/s41588-018-0048-5

Published Online: 2018-02-05

Published Print: 2018-03

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Scholl, Ute I. https://orcid.org/0000-0002-0309-8045
Stölting, Gabriel https://orcid.org/0000-0002-2339-0545
Schewe, Julia

Thiel, Anne https://orcid.org/0000-0002-9523-2351
Tan, Hua

Nelson-Williams, Carol https://orcid.org/0000-0003-0768-4751
Vichot, Alfred A.

Jin, Sheng Chih

Loring, Erin

Untiet, Verena https://orcid.org/0000-0002-1888-6378
Yoo, Taekyeong

Choi, Jungmin

Xu, Shengxin

Wu, Aihua

Kirchner, Marieluise

Mertins, Philipp

Rump, Lars C.

Onder, Ali Mirza

Gamble, Cory

McKenney, Daniel

Lash, Robert W.

Jones, Deborah P.

Chune, Gary

Gagliardi, Priscila

Choi, Murim https://orcid.org/0000-0002-9195-1455
Gordon, Richard

Stowasser, Michael

Fahlke, Christoph

Lifton, Richard P.
License Information

Text and Data Mining valid from 2018-02-05
More Information

Article History

Received: 8 June 2017

Accepted: 20 December 2017

First Online: 5 February 2018

Competing interests

: Heinrich Heine University Düsseldorf has filed a patent application: EP17209972, Diagnosis and Therapy of Primary Aldosteronism.