Bromberg, Kenneth D
Mitchell, Taylor R H
Upadhyay, Anup K
Jakob, Clarissa G
Jhala, Manisha A
Comess, Kenneth M
Lasko, Loren M
Li, Conglei
Tuzon, Creighton T http://orcid.org/0000-0001-9706-610X
Dai, Yujia
Li, Fengling
Eram, Mohammad S
Nuber, Alexander
Soni, Niru B
Manaves, Vlasios
Algire, Mikkel A
Sweis, Ramzi F
Torrent, Maricel
Schotta, Gunnar http://orcid.org/0000-0003-4940-6135
Sun, Chaohong
Michaelides, Michael R
Shoemaker, Alex R
Arrowsmith, Cheryl H
Brown, Peter J
Santhakumar, Vijayaratnam
Martin, Alberto
Rice, Judd C
Chiang, Gary G http://orcid.org/0000-0003-3004-7400
Vedadi, Masoud
Barsyte-Lovejoy, Dalia
Pappano, William N
Article History
Received: 27 May 2016
Accepted: 23 November 2016
First Online: 23 January 2017
Change Date: 31 January 2017
Change Type: Erratum
Change Details: In the version of this article initially published online, the second paragraph of the introduction contained two unnecessary commas, and "Suv4-20h1" was incorrectly listed in place of "Suv4-20h2" in one instance. The first two sentences of the description of mouse Suv4-20h expression should read: "Mouse Suv4-20h1 is ubiquitously expressed throughout embryogenesis and adult homeostasis, and mice that are homozygous null for this gene are perinatal lethal and have incomplete penetrance10. Suv4-20h2 expression, by comparison, is much less abundant during murine development. It is highly restricted in the adult, and <i>Suv4-20h2</i> homozygous null mice display no apparent defects." These errors have been corrected in the print, PDF and HTML versions of this article.
Competing interests
: This study was sponsored by AbbVie. AbbVie contributed to the study design, research, and interpretation of data, writing, reviewing, and approving the publication. K.D.B., A.K.U., C.G.K., M.A.J., K.M.C., L.M.L., Y.D., N.B.S., V.M., M.A., R.F.S., M.T., C.S., M.R.M., A.R.S., G.G.C. and W.N.P. were employees of AbbVie at the time of the study.