Crossmark

Shieldin complex promotes DNA end-joining and counters homologous recombination in BRCA1-null cells

Crossref DOI link: https://doi.org/10.1038/S41556-018-0140-1

Published: 2018-08

Update policy: https://doi.org/10.1007/SPRINGER_CROSSMARK_POLICY

Authors

Dev, Harveer http://orcid.org/0000-0003-2874-6894
Chiang, Ting-Wei Will

Lescale, Chloe

de Krijger, Inge

Martin, Alistair G.

Pilger, Domenic

Coates, Julia

Sczaniecka-Clift, Matylda

Wei, Wenming

Ostermaier, Matthias

Herzog, Mareike

Lam, Jonathan

Shea, Abigail

Demir, Mukerrem

Wu, Qian

Yang, Fengtang http://orcid.org/0000-0002-3573-2354
Fu, Beiyuan

Lai, Zhongwu

Balmus, Gabriel http://orcid.org/0000-0003-2872-4468
Belotserkovskaya, Rimma

Serra, Violeta http://orcid.org/0000-0001-6620-1065
O’Connor, Mark J.

Bruna, Alejandra

Beli, Petra http://orcid.org/0000-0001-9507-9820
Pellegrini, Luca http://orcid.org/0000-0002-9300-497X
Caldas, Carlos

Deriano, Ludovic http://orcid.org/0000-0002-9673-9525
Jacobs, Jacqueline J. L. http://orcid.org/0000-0002-7704-4795
Galanty, Yaron

Jackson, Stephen P. http://orcid.org/0000-0001-9317-7937
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Text and Data Mining valid from 2018-07-18
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Article History

Received: 27 April 2018

Accepted: 11 June 2018

First Online: 18 July 2018

Competing interests

: S.P.J. receives some research funding from AstraZeneca and is a named inventor on patents describing the use of PARP inhibitors in cancer therapy. V.S.’s laboratory receives research funding support from AstraZeneca. M.O. and Z.L. are employees and shareholders of AstraZeneca.