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Stochastic modeling of aging cells reveals how damage accumulation, repair, and cell-division asymmetry affect clonal senescence and population fitness

Crossref DOI link: https://doi.org/10.1186/s12859-019-2921-3

Published Online: 2019-07-15

Published Print: 2019-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Song, Ruijie

Acar, Murat
Funding

Funding for this research was provided by:

National Institute on Aging (1DP2AG050461-01)

National Institute of General Medical Sciences (1R01GM127870-01)
License Information

Text and Data Mining valid from 2019-07-15

Version of Record valid from 2019-07-15
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Article History

Received: 8 April 2019

Accepted: 29 May 2019

First Online: 15 July 2019

Ethics approval and consent to participate

: Not applicable.

: Not applicable.

: The authors declare that they have no competing interests.

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