Crossmark

Receptor-interacting protein kinase 1 is a key mediator in TLR3 ligand and Smac mimetic-induced cell death and suppresses TLR3 ligand-promoted invasion in cholangiocarcinoma

Crossref DOI link: https://doi.org/10.1186/s12964-020-00661-3

Published Online: 2020-10-09

Published Print: 2020-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Lomphithak, Thanpisit

Choksi, Swati

Mutirangura, Apiwat

Tohtong, Rutaiwan

Tencomnao, Tewin

Usubuchi, Hajime

Unno, Michiaki

Sasano, Hironobu

Jitkaew, Siriporn http://orcid.org/0000-0002-7931-2319
Funding

Funding for this research was provided by:

Research Grant for New Scholar by The Thailand Research Fund and Office of the Higher Education Commission (MRG6080130)

Grants for Development of New Faculty Staff, Ratchadaphiseksomphot Endowment Fund (DNS 61-004-37-001-2)

The Thailand Research Fund and the Medical Research Council (UK), Newton Fund (DBG 5980006 and MR/N01247X/1)

The Scholarship from the Graduate School, Chulalongkorn University to commemorate the 72nd anniversary of his Majesty King Bhumibol Aduladej (GCUGE12-2)

The 90th Anniversary Chulalongkorn University Fund (GCUGR1125613083M)

The Overseas Research Experience Scholarship for Graduate Students from the Graduate School and Grant for Join Funding (Ratchadaphiseksomphot Endowment Fund), Chulalongkorn University
License Information

Text and Data Mining valid from 2020-10-09

Version of Record valid from 2020-10-09
More Information

Article History

Received: 22 April 2020

Accepted: 10 September 2020

First Online: 9 October 2020

Ethics approval and consent to participate

: The study protocol was approved by IRB of Tohoku University School of Medicine, Sendai, Japan. Informed consent was obtained.

: Not applicable.

: The authors declare that they have no competing interests.

Document is current

Any future updates will be listed below