Crossmark

Mice defective in interferon signaling help distinguish between primary and secondary pathological pathways in a mouse model of neuronal forms of Gaucher disease

Crossref DOI link: https://doi.org/10.1186/s12974-020-01934-x

Published Online: 2020-09-07

Published Print: 2020-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Vardi, Ayelet

Ben-Dor, Shifra

Cho, Soo Min

Kalinke, Ulrich

Spanier, Julia

Futerman, Anthony H. http://orcid.org/0000-0003-0013-0115
Funding

Funding for this research was provided by:

Children's Gaucher Research Fund
License Information

Text and Data Mining valid from 2020-09-07

Version of Record valid from 2020-09-07
More Information

Article History

Received: 27 May 2020

Accepted: 19 August 2020

First Online: 7 September 2020

Ethics approval

: Mice were maintained under specific pathogen-free conditions and handled according to protocols approved by the Weizmann Institute Animal Care Committee according to international guidelines.

: Not applicable

: The authors declare that they have no competing interests.

Document is current

Any future updates will be listed below