H2AX phosphorylation and DNA damage kinase activity are dispensable for herpes simplex virus replication
Crossref DOI link: https://doi.org/10.1186/s12985-016-0470-1
Published Online: 2016-01-27
Published Print: 2016-12
Update policy: https://doi.org/10.1007/springer_crossmark_policy
Botting, Carolyn
Lu, Xu
Triezenberg, Steven J. http://orcid.org/0000-0003-4184-5221
Funding for this research was provided by:
Van Andel Research Institute (US)