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Unlocking thalidomide response as a hemoglobin F augmentation agent in transfusion-dependent β-thalassemia: the genetic impact of BCL11A, HBS1L-MYB, and XmnI polymorphism

Crossref DOI link: https://doi.org/10.1186/s13000-026-01797-5

Published Online: 2026-07-07

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Nazir, Iram

Siddiqi, Muhammad Younus Jamal

Wadood, Maeesa

Rizwan, Muhammad
License Information

Text and Data Mining valid from 2026-07-07

Accepted Manuscript valid from 2026-07-07
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Article History

Received: 15 January 2026

Accepted: 18 June 2026

First Online: 7 July 2026

Declarations

:

: Ethical approval was obtained from the Ethical Review Committee of Baqai Medical University, Karachi (Ref: BMU-EC/04-2022). Written informed consent was obtained from all participants or their legal guardians prior to enrollment.

: The authors declare no competing interests.

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