Crossmark

Neurons derived from sporadic Alzheimer’s disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation

Crossref DOI link: https://doi.org/10.1186/s13195-017-0317-z

Published Online: 2017-12-01

Published Print: 2017-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Ochalek, Anna

Mihalik, Balázs

Avci, Hasan X.

Chandrasekaran, Abinaya

Téglási, Annamária

Bock, István

Giudice, Maria Lo

Táncos, Zsuzsanna

Molnár, Kinga

László, Lajos

Nielsen, Jørgen E.

Holst, Bjørn

Freude, Kristine

Hyttel, Poul

Kobolák, Julianna

Dinnyés, András http://orcid.org/0000-0003-3791-2583
Funding

Funding for this research was provided by:

EU FP7, STEMMAD (PIAPP-GA-2012-324451)

EU FP7, EpiHealth (HEALTH-2012-F2-278418)

EU FP7, EpiHealthNet (PITN-GA-2012-317146)

Richter Gedeon Research Grant (RG-IPI-2013_TP7/026)

Innovation Fund Denmark (BrainStem 4108-00008B)

Innovation Fund Denmark (NeuroStem 4096-00001B)