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Specific disruption of Lnk in murine endothelial progenitor cells promotes dermal wound healing via enhanced vasculogenesis, activation of myofibroblasts, and suppression of inflammatory cell recruitment

Crossref DOI link: https://doi.org/10.1186/s13287-016-0403-3

Published Online: 2016-10-28

Published Print: 2016-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Lee, Jun Hee

Ji, Seung Taek

Kim, Jaeho

Takaki, Satoshi

Asahara, Takayuki

Hong, Young-Joon

Kwon, Sang-Mo
Funding

Funding for this research was provided by:

National Research Foundation (NRF-2015M3A9B4051053)

National Research Foundation (NRF-2015R1A5A2009656)

National Research Foundation (NRF-2014R1A1A2056907)

Korean Health Technology RND Project (HI15C0498)

Korean Health Technology RND Project (HI14C2069)

Korean government, Brain Busan 21 program and Brain Korea PLUS 21 program (BB21, BK PLUS 21)
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Text and Data Mining valid from 2016-10-28

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Article History

Received: 24 July 2016

Revised: 30 August 2016

Accepted: 1 September 2016

First Online: 28 October 2016

Updates are available Correction dated 2017-03-09

Click to view Correction: https://doi.org/10.1186/s13287-017-0493-6