Crossmark

Endogenous mouse huntingtin is highly abundant in cranial nerve nuclei, co-aggregates to Abeta plaques and is induced in reactive astrocytes in a transgenic mouse model of Alzheimer’s disease

Crossref DOI link: https://doi.org/10.1186/s40478-019-0726-2

Published Online: 2019-05-20

Published Print: 2019-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Hartlage-Rübsamen, Maike

Ratz, Veronika

Zeitschel, Ulrike

Finzel, Lukas

Machner, Lisa

Köppen, Janett

Schulze, Anja

Demuth, Hans-Ulrich

von Hörsten, Stephan

Höfling, Corinna

Roßner, Steffen https://orcid.org/0000-0002-9959-283X
Funding

Funding for this research was provided by:

Bundesministerium für Bildung und Forschung (01ED1501A, 01ED1501B, 01ED1501C)

Alzheimer Forschung Initiative (AFI #16004)

Deutsche Forschungsgemeinschaft (Open Access Publishing)
License Information

Text and Data Mining valid from 2019-05-20

Version of Record valid from 2019-05-20
More Information

Article History

Received: 16 April 2019

Accepted: 17 April 2019

First Online: 20 May 2019

Ethics approval and consent to participate

: All applicable international, national, and/or institutional guidelines for the care and use of animals were followed. All experiments carried out were in agreement with European regulations on the protection of animals used for scientific purposes (Directive 2010/63/EU), and they were also approved by the Ethical Committee for Animal Research of Landesdirektion Sachsen, license numbers T11/16, T14/16 and T28/16.

: All the authors have approved publication.

: The authors declare that they have no competing interests.

: Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.

Document is current

Any future updates will be listed below