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Mice deficient in the C-terminal domain of TAR DNA-binding protein 43 develop age-dependent motor dysfunction associated with impaired Notch1−Akt signaling pathway

Crossref DOI link: https://doi.org/10.1186/s40478-019-0776-5

Published Online: 2019-07-25

Published Print: 2019-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Nishino, Kohei

Watanabe, Seiji

Shijie, Jin

Murata, Yuri

Oiwa, Kotaro

Komine, Okiru

Endo, Fumito

Tsuiji, Hitomi

Abe, Manabu

Sakimura, Kenji

Mishra, Amit

Yamanaka, Koji http://orcid.org/0000-0003-4655-0035
Funding

Funding for this research was provided by:

Japan Society for the Promotion of Science (18H04860, 17H04986)

Uehara Memorial Foundation (none)
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Text and Data Mining valid from 2019-07-25

Version of Record valid from 2019-07-25
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Article History

Received: 27 June 2019

Accepted: 18 July 2019

First Online: 25 July 2019

Ethics approval and consent to participate

: All the experiments were conducted in compliance with the ARRIVE guidelines. The experiments using genetically modified mice were approved by the Animal Care and Use Committee and the recombinant DNA experiment committee of Nagoya University (approval numbers #19269 and #143, respectively).

: The authors declare that they have no competing interests.

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