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TDP-43 pathology is sufficient to drive axon initial segment plasticity and hyperexcitability of spinal motoneurones in vivo in the TDP43-ΔNLS model of Amyotrophic Lateral Sclerosis

Crossref DOI link: https://doi.org/10.1186/s40478-025-01934-z

Published Online: 2025-02-24

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Djukic, Svetlana

Zhao, Zhenxiang

Jørgensen, Lasse Mathias Holmsted

Bak, Anna Normann

Jensen, Dennis Bo

Meehan, Claire Francesca
Funding

Funding for this research was provided by:

China Scholarship Council

Independent Research Fund Denmark

The Lundbeck Foundation (R370-2021-1109)

Copenhagen University
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Text and Data Mining valid from 2025-02-24

Version of Record valid from 2025-02-24
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Article History

Received: 12 October 2024

Accepted: 24 January 2025

First Online: 24 February 2025

Declarations

:

: This animal research was granted by the Danish Animal Experiments Inspectorate (authorization no. 2018-15-0201-01426), and all treatments conformed to the national and European laws on the welfare and protection of animals used for experimentation - i.e., the Danish Animal Welfare Act (2013) and the EU directive 2010/63/EU.

: Not applicable.

: The authors declare no competing interests.

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