Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite
Crossref DOI link:
Update policy: https://doi.org/10.1007/SPRINGER_CROSSMARK_POLICY
Garfield, Alastair S.
Davies, Jennifer R.
Burke, Luke K.
Furby, Hannah V.
Wilkinson, Lawrence S.
Heisler, Lora K.
Isles, Anthony R.
Funding for this research was provided by:
Biotechnology and Biological Sciences Research Council (BB/J016756/1)
Medical Research Council (G0801418)
Foundation for Prader-Willi Research (n/a)