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Histone hypo-acetylation of Sox9 mediates nicotine-induced weak cartilage repair by suppressing BMSC chondrogenic differentiation

Crossref DOI link: https://doi.org/10.1186/s13287-018-0853-x

Published Online: 2018-04-10

Published Print: 2018-12

Update policy: https://doi.org/10.1007/springer_crossmark_policy

Authors

Tie, Kai

Wu, Min

Deng, Yu

Wen, Yinxian

Dan Xu,

Chen, Liaobin

Wang, Hui
Funding

Funding for this research was provided by:

National Natural Science Foundation of China (81371940, 81220108026, 81702159)

Hubei Province health and family planning scientific research project (WJ2017Q012)
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Article History

Received: 27 December 2017

Revised: 2 March 2018

Accepted: 21 March 2018

First Online: 10 April 2018

Ethics approval

: All animal experiments were performed in accordance with the Guidelines for the Care and Use of Laboratory Animals from the National Research Council of the United States National Academies, and approved by the Committee on the Ethics of Animal Experiments of the Wuhan University School of Medicine.

: Not applicable.

: The authors declare that they have no competing interests.

: Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.