Crossmark

Cancer‐associated POT1 mutations lead to telomere elongation without induction of a DNA damage response

Crossref DOI link: https://doi.org/10.15252/embj.2020107346

Published Online: 2021-05-02

Published Print: 2021-06-15

Update policy: https://doi.org/10.1002/crossmark_policy

Authors

Kim, Won‐Tae https://orcid.org/0000-0002-9359-3073
Hennick, Kelsey https://orcid.org/0000-0002-6127-2400
Johnson, Joshua https://orcid.org/0000-0003-0995-9894
Finnerty, Brendan

Choo, Seunga

Short, Sarah B

Drubin, Casey

Forster, Ryan

McMaster, Mary L

Hockemeyer, Dirk https://orcid.org/0000-0002-5598-5092
Funding

Funding for this research was provided by:

American Cancer Society (133396‐RSG‐19‐029‐01‐DMC)

Pew Charitable Trusts

Alexander and Margaret Stewart Trust
License Information

Version of Record valid from 2021-05-02
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Publication History

Received: 2020-11-18

Accepted: 2021-03-16

Published: 2021-05-02